A complete yet brief lecture about the Respiratory system. This Lecture focuses on the Anatomy and Physiology, Diagnostic Exams, Lab Values, Respiratory Diseases and Nursing Managements.
Brief Review of System Nursinglectures.blogspot.com
Trachea – slightly movable & quickly returns to midline after displacement
Tactile fremitus –transmission of vibration of air movement through chest wall during phonation (99 method)
Thoracic excursion
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Percussion:
Resonant – low-pitched hollow (normal lung sound)
Hyperresonant – louder & lower-pitched; presence of increased amount of air (emphysema, pneumothorax)
Dull- thudlike
Tympanic – hollow (tension-pneumothorax)
Flat – soft high-pitched
Auscultation:
Bronchial, bronchovesicular, vesicular
Adventitious Breath sounds:
Stridor - High pitched crowing sound, usually heard on inspiration, indication of a tight upper airway
Wheezing - Whistling sound, usually heard on expiration, indication of narrowing of lower airways (bronchospasm, edema, foreign material)
Ronchi - Rattling sound, caused by mucus in larger airways
Rales - Fine crackling sound, indication of fluid in the alveoli
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Diagnostics
Chest X-ray (Chest radiography; Serial chest x-ray)
Visualization of the chest, lungs, heart, large arteries, ribs, and diaphragm while standing in front of the machine
Two views are usually taken:
Antero-posterior view - x-rays pass through the chest from the back
Lateral view - x-rays pass through the chest from one side to the other
Nursing Interventions:
Instruct client to hold his breath while x-ray is taken
Inform client that test is performed in the radiology department (in hospitals, mobile x-rays may be used) & the film plate may feel cold
Instruct client to wear a hospital gown and remove all jewelries
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B. Pulmonary Function Tests (PFT)
a group of tests measuring lung function
Measure of diffusion capacity
client breathes in a harmless gas for a very short time (one breath)
the concentration of the gas in the air exhaled is measured
the difference in the amount of gas inhaled and exhaled can help estimate how quickly gas can travel from the lungs into the blood
Body plethysmograph - most accurate
Client sits in a sealed, clear box that looks like a telephone booth while breathing in and out into a mouthpiece
Changes in pressure inside the box help determine the lung volume
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Cont…(PFT)
Spirometry test – measures airflow; client will breathe through a tight fitting mouthpiece and will have nose clips
Nursing Interventions: Instruct client to:
breathe into a mouthpiece that is connected to an instrument (spirometer)
eat a light meal before the test
not to smoke for 4 - 6 hours before the test
stop using bronchodilators or inhaler medications 6-8hrs prior
Inform client that temporary shortness of breath or light-headedness may be felt
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C. Peak Expiratory Flow Rate (PEFR)
measures how fast a person can exhale
it is one of many tests that measure how well the airways work
requires a peak expiratory flow (PEF) monitor, a small handheld device with a mouthpiece at one end and a scale with a moveable indicator (usually a small plastic arrow)
commonly used to diagnose and monitor lung diseases such as asthma, chronic bronchitis, chronic obstructive pulmonary disease (COPD), & emphysema
Home monitoring helps determine whether treatments are working or detect when your condition is getting worse . This allows anticipation on when breathing will bec ome worse and to take medications or to call hea lth care providers before symptoms become too seve re
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A decrease in peak flow indicates blocked or narrowed airways
A significant fall in peak flow can signal the onset of a lung disease esp. when accompanied by persistent coughing, SOB, or wheezing
PEFR measurements are not as accurate as the spirometry
Nursing Interventions:
Inform client that repeated efforts may cause lightheadedness
Loosen any tight clothing that might restrict breathing
Sit up straight or stand while performing the tests
Instruct client on proper procedure to do this test:
Breathe in as deeply as possible.
Blow into the instrument's mouthpiece as hard and fast as possible.
Do this 3 times, and record the highest flow rate
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D. Throat Culture
Also known as throat swab culture
a laboratory test to isolate and identify organisms that may cause infection in the throat; when throat infection is suspected, particularly strep throat
back of the throat is swabbed with a sterile cotton swab near the tonsils
Nursing Interventions:
Instruct client not to use antiseptic mouthwashes before the test
Inform client that he may experience a gagging sensati on when the back of the throat is swabbed
Instru ct to resist gagging and closing the mouth during procedure (test only takes a few seconds)
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E. Bronchoscopy (Fiber Optic Bronchoscopy)
views the airways and diagnose lung disease
may also be used during the treatment of some lung conditions
flexible bronchoscope is usually used (less than ½ in wide and about 2ft long)
scope is passed through the mouth or nose, and then into the lungs
rigid bronchoscope requires general anesthesia
flexible bronchoscope uses local anesthesia (spray if via mouth and throat; numbing jelly if via nose)
IV meds may be given to help relax the client
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Cont…(Bronchoscopy)
Nursing Interventions:
Inform client that spraying of local anesthesia will cause coughing at first, which will stop as the anesthetic begins to work
Inform client that as the anesthesia wears off, the throat may be scratchy for several days
Instruct client on NPO 6-12hrs prior (withhold ASA or Ibuprofen if client takes it on a regular basis or as ordered)
Place client on NPO 1-2hrs after the procedure or until (+) for gag reflex
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F. Sputum Culture
Sputum - secretion produced in the lungs and the bronchi; what comes up with deep coughing
This mucus-like secretion may become infected, bloodstained, or contain abnormal cells that may lead to a diagnosis
Nursing Interventions:
Drinking a lot of water and other fluids the night before collection may help
Perform back tapping or chest clapping on client to aid in loosening the sputum
Instruct client on proper specimen collection
Collect morning specimen
Gargle with water only before specimen collection cough deeply and spit sputum in a sterile cup
Send specimen to lab ASAP
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G. Oximetry
measures oxygen concentration (%) in the blood
used in the evaluation of various medical conditions affecting heart & lung functions
most commonly used = pulse oximeters because they respond only to pulsations, such as those in pulsating capillaries of the area tested
pulse oximeter works by passing a beam of red and infrared light through a pulsating capillary bed
ratio of red to infrared blood light transmitted gives a measure of the oxygen saturation in the blood
Principle: oxygenated blood is bright red while the deoxygenated blood is blue-purple
Other types:
intracardiac oximetry - blood that is within the heart or on whole blood that has been removed from the body
More recently, using a similar technology to oxymetry, carbon dioxide levels can be measured at the skin as well
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Pulmonary Tuberculosis
contagious bacterial infection that mainly involves the lungs, but may spread to other organs
Cause: Mycobacterium tuberculosis
Mode of transmission: inhalation of air droplets from a cough or sneeze of an infected person
primary stage of the infection is usually asymptomatic
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High-risk individuals
Elderly
Infants
Immunosuppressed (AIDS, chemotherapy, or antirejection medicines given after a organ transplant)
Are in frequent contact with people who have the disease
Live in crowded or unsanitary living conditions
Have poor nutrition
The appearance of drug-resistant strains of TB
S/Sx
Limited to minor cough
Fever and night sweats
Fatigue
Unintentional weight loss
Excessive sweating, especially at night
Coughing up blood
Phlegm-producing cough
Wheezing
Chest pain
Breathing difficulty
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Cont…(PTB)
Dx:
Chest x-ray – seen on upper lobes (due to higher O 2 concentration)
Sputum cultures (Acid-Fast Stain) – confirmatory test
Tuberculin skin test (Mantoux Test) – ID purified protein derivative (PPD)
48-72hrs interpretation
(+) = 15mm induration (5mm for immunosuppressed clients)
Bronchoscopy
Thoracentesis (very rare occasions)
Chest CT Scan
Complications:
Miliary TB - widespread dissemination of Mycobacterium tuberculosis from hematogenous spread
Pleural Effusion – collection of fluid in the pleural cavity
Empyema – purulent drainage It results from an untreated pleural-space infection
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Cont…
Tx: Multi-drug therapy = to prevent development of resistance ( RIPES )
R ifampicin – inhibits RNA synthesis of the bacilli
I soniazid – remarkably potent to the bacilli; prophylaxis; given with Vit. B 6
P yrazinamide (PZA) – inhibits cell growth
E thambutol – inhibits cell growth
S treptomycin – 1 st drug found to be effective against PTB; given by injection
Nursing Management:
Give meds before meals
Maintenance therapy = after 6months
Client not communicable after 2wks
Rifampicin’s SE: reddish/orange body secretions (urine)
PZA prone to hyperuricemia so ↑ oral fluids
Ethambutol - A/E: optic neuritis so √ vision/visual changes
C/I: pedia – cannot report any visual disturbances
Streptomycin – A/E: ototoxic (√ tinnitus)
nephrotoxic = √ oliguria
neurotoxic = seizure precautions
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Asthma
Chronic inflammatory airway disease
Exposure to allergens (dust, smoke,
animal dander, pollen, volatile organic
compounds, food, meds, etc)
Cold air, exercise, & emotional upset
can produce bronchospasm
Pathophysiology:
allergens -> immune response (mast cells, eosinophils, T lymphocytes) -> mucus production -> bronchospasm -> inflammation -> excessive mucus production -> narrowing of airways -> bronchoconstriction -> asthma attack
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Manifestations: (asthma attacks differ from 1
person to another)
Episodic wheezing
Feelings of chest tightness
Cough may be accompanied by wheezing
Prolonged expiration
Increased RR
Severe attacks = severe dyspnea (use of accessory muscles)
Distant breath sounds (due to air trapping)
Loud wheezing
Fatigue develops
Moist skin
Anxiety/panic attack
Client is able to speak 1-2 words before taking a breath
Inhalation challenge test – measures the level of airway responsiveness (histamine, or exposure to non-pharmacologic agent)
Tx/ Nursing Management: goal = prevention of attack episodes
Pharmacologic
Quick-relief – not for daily use; relaxes bronchial muscles (albuterol, terbutaline via MDI or nebulizer)
Long-term meds – taken on daily basis; anti-inflammatory (cromolyn via MDI), corticosteroids (budesonide via MDI), bronchodilators (theophylline)
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Mgt:
B ronchodilators
R est & relaxation techniques
O 2 = low flow (1-2Lpm)
N ebulize
C hest physiotherapy & controlled breathing (IPPB)
H igh-fowler’s/ orthopneic
I mmunotherapy
A void allergens
L iberal fluid intake
Meds:
A minophylline
S teroids
T heophylline – relaxes bronchial muscles
H istamine antagonist
M ucolytics – acetylcysteine (Fluimucil)
A ntibiotics
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Chronic Obstructive Pulmonary Disease (COPD)
clinical syndrome of chronic dyspnea as a result of expiratory airflow obstruction due to chronic bronchitis or emphysema (often both)
Causes: long-term smoking (leading cause) & Alpha1-antitrypsin deficiency (only known inherited form of the disease)
Risk factors:
Exposure to certain gases or fumes in the workplace
Exposure to heavy amounts of second hand smoke and pollution
Frequent use of cooking gas without proper ventilation
Low socioeconomic status
Male
Living in heavily industrialized urban areas
Recurrent respiratory illnesses
Family history of chronic bronchitis and emphysema (e.g., alpha1-antitrypsin deficiency)
Emotional stress and repressed emotions have also been shown to contribute
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Chronic Bronchitis (“Blue Bloaters”)
chronic cough, resulting from excessive tracheobronchial mucus production and impaired mucus elimination, on most days for 3 months of a year, for 2 consecutive years
Some people, even those with severe COPD, have few or no symptoms
Pathophysiology:
hallmarked by hyperplasia (increased number) and hypertrophy (increased size) of the goblet cells (mucous gland) of the airway -> increase mucus secretion -> airway obstruction -> cyanosis
infiltration of the airway walls with inflammatory cells (neutrophils) -> scarring -> airway wall thickening -> narrowing of the small airway -> metaplasia (abnormal change in the tissue) & fibrosis (further thickening and scarring) of lower airway -> limitation of airflow -> cyanosis
enlarged air spaces distal to the terminal bronchioles with destruction of the alveolar walls; there is also a loss of elastic recoil in the lung
Pathophysiology:
exact mechanism for the development of emphysema is not understood, although it is known to be linked with smoking and age
enlarged air sacs (alveoli) of the lungs -> reduces lung surface area -> ↓ lung elasticity -> small bronchioles collapse -> dead air space formation (blebs) -> air trapping -> dyspnea
Meds: bronchodilators - to increase airflow and reduce dyspnea
sometimes theophylline - requires frequent blood monitoring for toxicity
inhaled steroids
Antibiotics - during flare-ups of symptoms
Alpha1-antitrypsin replacement therapy
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Pleurisy
inflammation of the lining of the lungs that ca uses pain when you take a breath or cough
normally smooth lining of the lungs (the pleura) become rough, they rub together with each breath, and may produce a rough, grating sound called a "friction rub."
Causes:
may develop when you have lung inflammation due to infections such as pneumonia or tuberculosis
Asbestos-related disease
Certain cancers
Chest trauma
Pulmonary embolus - blockage of an artery in the lungs by fat, air, blood clot, or tumor cells
Respiratory tract infections
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S/Sx: main symptom = chest pain
Some people feel the pain in the shoulder
Deep breathing, coughing, and chest movement makes the pain worse
fluid may collect inside the chest cavity & may cause the following:
Thoracentesis - procedure to remove fluid from the space between the lining of the outside of the lungs (pleura) and the wall of the chest; local anesthesia
Pleural Biopsy - procedure to remove a sample of the tissue lining the lungs and the inside of the chest wall to check for disease or infection
Ultrasound of the chest or Chest x-ray
Sputum exam
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Tx: depends on what is causing the pleurisy
Bacterial infections = antibiotics (some bacterial infections require a surgical procedure to drain all the infected fluid)
acetaminophen or anti-inflammatory drugs such as ibuprofen (for pain control)
Thoracentesis
Complications: Collapsed lung due to thoracentesis
Complications from the original illness
Nursing Management:
Health teachings (infection, work environment, splinting ribcage with pillow)
Position client on affected side
Thoracentesis: Instruct client not to cough, breathe de eply, or move during the test to lung puncture
Instruct to report SOB &/or chest pain during procedure
Apply pressure on puncture site & monitor for bleeding
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Tracheostomy
Tracheostomy – used for severe lung disorder , neurological problem, or infection makes it impossible to breathe,
to keep the windpipe open and supply air
a small opening (stoma) through the skin on the throat
a breathing tube is directly inserted into the windpipe (trachea).
The trache tube is sometimes sewn to the skin around the stoma
It can also be held in place with trache ties
Some trache tubes have an inflatable cuff near the outer end to keep it from coming out and to prevent air leaks
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trache tube parts
Obturator - used to pass the trache into the windpipe
outer cannula (tube) - has a plastic "trache plate" that lies against the skin of the neck and holds the trache in place
Inner cannula that fits inside the outer one and locks into place
Obturator and clamp should always be at bedside
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Tracheostomy Care
clean the inner cannula on a daily basis
Observe proper precautions & handwashing before & after care
Whenever the tube threatens to become clogged with mucus, suction it clear
Materials:
kidney basin
a small brush (like a toothbrush) or twisted OS
H 2 O 2 &/or sterile NSS
4x4 gauze pad
scissors
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Procedures:
Place a “trache bib” under the trache plate with a gauze pad (upright “U”)
Unlock the inner cannula and remove it by pulling it gently out and down
Put a clean wet inner cannula (if reserve is available) as replacement & lock in place
Clean the dirty cannula by soaking it in H 2 O 2
Scrub it with the small brush when bubbling stops
Rinse well the inner cannula by pouring the sterile NSS
Return in place & lock if client has no reserve
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Endotracheal (ET) Tube
most common artificial airway used for short-term airway management or mechanical ventilation
may be inserted either orally or nasally
has a cuff that is inflated with air to hold the tube in place in the trachea
amount of air in the cuff should be checked every 8hrs to ensure that the cuff is not exerting too much pressure on the trachea walls
client with ET tube must be closely monitored:
to ensure that the tube remains patent
that skin breakdown does not occur from the tube (either the oral or nasal cavity)
infection is prevented
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Intubation Illustrations Nursinglectures.blogspot.com 2 Intubation 3 ET tube Placement 4 Securing the ET tube 1 ET tubes
Securing Apparatuses for ET Tube Nursinglectures.blogspot.com ETAD Thomas Tube Holder
Nursing Management
RNs prepare all needed materials needed for in tubation &/or assist in placement by securing p at ient’s position (head tilted on supine)
Sterile suction kit, a bottle of sterile NSS, sterile gloves, a clean bite block if necessary, and tape already torn into appropriately-sized pieces, laryngoscope
Documentation (note also tube distance at client’s lips)
All waste should be properly disposed
Complete airway check every 8hrs & prn
The insertion point (in cm) of the ET tube should be confirmed to be the same as prior to the procedure, unless the purpose of the procedure was to change the depth of the tube (via X-ray)
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Cont…
Primary portion of ET tube management is suctioning every 2hrs or prn
Client should be hyperoxygenated prior to suctioning
Color and amount of any sputum return should be noted
Oral cavity should also be suctioned
Thorough oral care every 8hrs and prn
If client has a bite block, it must be removed and cleaned or replaced every 8hrs
tube should be repositioned so as not to continuously exert pressure in the same area
If the tube is taped to the client's face, tape must be removed and replaced on the opposite side of the face at least once per day and prn
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Devices for Oxygen Administration
nasal cannula (NC) - thin tube with two small nozzles that protrude into the nostrils
It can only provide oxygen at low flow rates, 2-6 litres per minute (LPM), delivering a concentration of 28-44%.
simple face mask - basic mask used for non-life-threatening conditions but which may progress in time
Often set to deliver oxygen between 2-10 LPM
The final oxygen concentration delivered by this device is dependent upon the amount of room air that mixes with the oxygen
non-rebreather mask- utilized for those requiring high-flow oxygen, but do not require breathing assistance
It has an attached reservoir bag where oxygen fills in between breaths, and a valve that largely prevents the inhalation of room or exhaled air.
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Cont…
bag-valve-mask (Ambubag) - use d in CPR or if client is in critical condition
An oxygen reservoir bag is attached to a central cylindrical bag, attached to a valved mask
administers almost 100% concentration oxygen at 8-15 Lpm
The central bag is squeezed manually to deliver a "breath"
anaesthetic machine - used for general anesthesia
allows a variable amount of oxygen to be delivered, along with other gases including air, nitrous oxide and inhalational anaesthetics
Before trying to study a particular system, it is very important to have a good lecture on the anatomy and physiology.
In this nursing lecture, you will be able grasp the lessons easily and adapt it in your nursing study.
Outline
Structure and Function
Subjective Data
Objective Data
Abnormal Findings
Structure and Function
Thoracic Cage /Cavity
Shape- bony, conical shape, narrower at top borders – it is defined by:
Sternum – 3 parts: manubrium, body, xiphoid process
Ribs – 12 pairs, 1 st seven attach to the sternum (costal cartilages) Ribs 8,9,&10 attach to the costal cartilage above, Ribs 11 & 12 are floating ribs
12 Thoracic vertebrae
Diaphragm – the floor, separates the thoracic cavity from the abdomen
Anterior Thoracic Landmarks
Suprasternal Notch – U shaped depression
Sternum – “breastbone” = 3 parts
Manubrium
Body
Xiphoid process
Angle of Louis – manubriosternal angle continuous with the 2 nd Rib
Costal angle- usually 90 0 or <. (increases when rib cage is chronically overinflated)
Posterior Thoracic Landmarks
Vertebra Prominens – Flex head, feel most prominent bony projection at base of neck = C7 next lower one is T1
Spinous Processes – spinal column-
Scapula – symmetrical , lower tip at the 7 -8 th Rib
12 th Rib = midway b/t spine & side
Reference Lines
Anterior Chest
Midsternal line
Midclavicular line
Posterior Chest
Vertebral line – midspinal
Scapular line
Lateral Chest
Anterior Axillary line
Posterior Axillary line
Mid–axillary line
The Thoracic Cavity
Mediastinum middle of the thoracic cavity & contains;
Esophagus
Trachea
Heart
Great Vessels
Pleural Cavities on either side of the mediastinum contain the lungs
Lung Borders
Anterior Chest –
Apex 3 -4 cm. ↑ inner 1/3 of the clavicles
Base – rests on the diaphragm, 6 th rib, MCL
Lateral Chest
Extends from Axilla apex to 7 th –8 th rib
Posteriorly
Apex of lung is at C7 – Base T10 (on deep inspiration to T12)
Lobes of Lung
Right Lung
3 lobes, upper, middle , lower
Shorter due to liver
Left Lung
LUL = Left Upper and Lower ( 2 lobes)
Narrower due to heart
Lobes
Diagonal sloping segments
Oblique fissures
3 Important Points
Left Lung – no middle lobe
Anterior chest contains upper & middle lobes with very little lower lobe
Posterior chest has almost all lower lobe. Rt middle lobe does not project into the posterior chest
Pleurae
The Pleurae form an envelope b/t the lungs & chest wall
Visceral pleura – lines outside of lungs
Parietal pleura – lines inside of chest wall & diaphragm
Pleural Cavity – the inside of the envelope- space b/t visceral & parietal pleura, lubrication. Normally has a vacuum or neg. pressure
Tracheal & Bronchial Tree
Trachea – anterior to esophagus-
10-11 cm.long, begins at cricoid cartilage
Bifurcates just below the sternal angle ( AKA angle of Louis, manubriosternal angle) into the
Right Main Stem Bronchus – shorter, wider, more vertical ( Intubation – listen to breath sounds bilaterally)
Left Main Stem Bronchus
Tracheal & Bronchial Tree
The trachea & bronchi provide the passage for air to get into the lungs from the environment = Dead Space (no air exchange takes place here)
Bronchi
Secrete mucus – captures particles
Cilia – moves the trapped particles up to be expelled or swallowed
Pleurisy, pleural thickening, pneumothorax (air), pleural effusion (fld.) in the pleural space
Increased Breath Sounds = dense lung tissue enhances sound transmission as in consolidation ie. pneumonia
Silent chest = ominous
Adventitious Sounds
Not normally heard in the lungs. Caused by moving air colliding with secretions or by popping open of previously deflated airways
Crackles (Rales)
Fine – high pitched popping- not cleared by coughing. Simulate sound by rolling strand of hair b/t fingers near ear or moisten thumb& index finger & separate them near your ear
Course crackles- (opening a velcro fastener)
Pleural Friction Rub – coarse & low pitched, 2 pieces of leather rubbed together close to ear
Adventitious Sounds
Wheeze (Rhonchi)
High pitched, musical squeaking = air squeezes - asthma
Stridor – high pitched, inspiratory, crowing, louder in neck = croup, acute epiglottitis
Coarse Crackles
Fine Crackles
Voice Sounds normal voice transmission is soft, muffled & indistinct. Pathology that ↑ lung density makes words clearer
Bronchophony – “99”
Egophony- ee-ee-ee if disease sounds like aa-aa-aa Record as “E -> A changes”
Whisper pectoriloquy 1-2-3
These tests are only done if lung pathology is suspected
Anterior Chest
Inspect
Shape & Configuration
Expression- relaxed
LOC – alert & cooperative
Skin color & condition
Quality of Respirations – reg. & even, no retraction or use of accessory muscles
Anterior Chest
Palpate
Symmetric Chest Expansion
Tenderness, turgor, temp., moisture
Tactile Fremitus
Compare both sides
Symmetric Expansion
Sequence for percussion & auscultation
Tactile fremitus
Percussion
Apices in Supraclavicular Areas
Interspaces = Resonance
Dullness
Female breast tissue
Liver – Rt. 5 th intercostal space midclavicular
Heart – Lt. 3 rd intercostal space midclavicular
Flat = muscle & bone
Tympany = stomach (Lt. Side)
Expected Percussion Notes
Auscultate
Apices (supraclavicular) to 6 th rib
Bilateral moving down
One full respiration
Directly over chest wall – displace female breast tissue
Location Of Breath Sounds
Pulse Oximeter
Noninvasive measurement of arterial oxygen saturation = SpO 2 by measuring the relative amt. of light absorbed by oxyhemoglobin and unoxygenated hemoglobin. It compares light emitted to amt absorbed. Normally 97 -98%
Terms for Documentation
Rate
Eupnea 12 – 20 bpm normal
Tachypnea > 24, rapid, shallow
Bradypnea <>
Apnea = No respirations for 10 sec. or more
Pattern = breathing rhythm. Normal respirations are regular and even.
Cheyne – stokes = resp wax & wane in reg pattern with periods of apnea(20sec)
Biot’s or ataxisic Sim. To cheyne –stokes but pattern irreg.
Depth – on inspiration the normal depth is nonexaggerated and effortless.
Shallow
Sighing – purposeful to expand the alveoli
Symmetry – bilateral rise and fall of the chest with respiration
Audibility – normally be heard by the unaided ear several centimeters from the patient’s nose/mouth
Patient position – healthy person breathes comfortably in supine, prone or upright position
Orthopnea
Mode of Breathing – normally inhale/exhale through nose
Hi, This is a new Nursing lecture about Mechanical Ventilation. What is Mechanical Ventilation? In this Nursing lecture, you will know what are the indications for mechanical ventilation.
Indications for Mechanical Ventilation
Impending Respiratory Failure
Acute Respiratory Failure/Arrest
Post-Operatively
Indications for Mechanical Ventilation
Impending Respiratory Failure
Progressively worsening clinical appearance.
Worsening CXR.
Hypoxemic Respiratory Failure.
Hypercapnic Respiratory Failure.
Indications for Mechanical Ventilation
Acute Respiratory Failure/Arrest
Acute change in ABG results
Respiratory Arrest/Status Post CPR
Acute epiglottitis/anaphylaxis
Indications for Mechanical Ventilation
Postoperatively
Oversedation/paralytics
Pain Control
Proper Immobilization
Key Terms in Mechanical Ventilation
Tidal Volume (Vt)
The volume of air inhaled and exhaled from the lungs.
Breaths per Minute (RR, f)
Also known as frequency.
Positive End Expiratory Pressure (PEEP)
Maintenance of above atmospheric pressure at the airway throughout exp. phase.
Key Terms in Mechanical Ventilation
Minute Ventilation (VE)
The total amount of volume moving in and out of the lung in one minute.
Fractional Inspired Oxygen (FiO2)
Correctly written with decimal place (21%-0.21; 100%-1.0)
Inspiratory:Expiratory Ratio (I:E ratio)
Normal I:E ratio 1:2-3
Negative Pressure Ventilation
Rarely Used; Currently used for patients with neuromuscular diseases.
Thoracic cage is encased where negative pressure is applied across the chest wall.
Generates subatmospheric pressures creating a difference in pressure gradients.
During exhalation, negative pressure is replace by atmospheric pressure allowing the lungs to deflate.
Negative Pressure Ventilation
Types of Negative Pressure Ventilators
Iron Lung circa 1950’s
Modern(ized) Iron Lung
Chest Cuirass
Complications with Negative Pressure Ventilation
Limited access for patient care.
Inability to properly monitor pulmonary mechanics.
Patient discomfort.
Positive Pressure Ventilation
Defined as the application of pressure to the lungs in order to improve gas exchange.
The Lungs are physically filled/ventilated with air using machinery.
Multiple modes, methods, and theory.
Positive Pressure Ventilation
Basically broken into two categories:
Control Modes.
Supportive Modes.
Control Modes of Ventilation
Assist/Control (usually abbreviated A/C also known as Volume Control VC).
Tidal Volume is set and remains constant.
Respiratory Rate is set.
Airway Pressure will vary according to lung compliance.
Ventilator will deliver set volume whether patient triggers a breath or mandatory breath is being delivered.
Control Modes of Ventilation
Pressure Control Ventilation (usually abbreviated PCV or sometimes PCIRV).
Upper Airway Pressure Level is set and remains constant.
Respiratory Rate is set.
Tidal volumes will vary according to lung compliance.
Ventilator will deliver set pressure level whether patient triggers a breath or mandatory breath is being delivered.
Control Modes of Ventilation
Pressure Regulated Volume Control (usually abbreviated PRVC).
Tidal Volume is set, however may or may not remain constant.
Respiratory Rate is set.
Ventilator will deliver volume however volume may decrease according to patient’s lung compliance.